init write with jabref

* diff with this commit shows just three small changes made when
initially opening and re-writing the omega.bib bibtex file with jabref.
  * jabref added a utf encoding msg on first line
  * jabref added a short config comment on last line
  * converted and rewrote fields for an old Seung:2000 reference---this
  one had a field called 'ps' (something inherited from endnote/medline
  import 18 years ago!) which jabref doesn't like as ps and pdf are old
  deprecated field names for external files. So that field was changed
  slightly by jabref. I also changed the Bdsk-Url-1 reference to a doi
  field for jabref to use.

OMEGA.bib

@@ -1,3 +1,4 @@
+% Encoding: UTF-8
 @article{Belanger:2011a,
 	Abstract = {The energy requirements of the brain are very high, and tight regulatory mechanisms operate to ensure adequate spatial and temporal delivery of energy substrates in register with neuronal activity. Astrocytes-a type of glial cell-have emerged as active players in brain energy delivery, production, utilization, and storage. Our understanding of neuroenergetics is rapidly evolving from a "neurocentric" view to a more integrated picture involving an intense cooperativity between astrocytes and neurons. This review focuses on the cellular aspects of brain energy metabolism, with a particular emphasis on the metabolic interactions between neurons and astrocytes.},
 	Author = {B{\'e}langer, Mireille and Allaman, Igor and Magistretti, Pierre J},
@@ -32517,42 +32518,41 @@ CONCLUSIONS: Centrifugal axons in the macaque retina are part of the system of a
 	File = {papers/Shu_Nature2003.pdf},
 	Bdsk-Url-1 = {http://dx.doi.org/10.1038/nature01616}}
 
-@article{Seung:2000,
+@Article{Seung:2000,
-	Address = {Howard Hughes Medical Institute and Department of Brain and Cognitive Sciences, Massachusetts Institute of Technology, Cambridge 02139, USA. seung@mit.edu},
+  author        = {Seung, H S},
-	Author = {Seung, H S},
+  title         = {Half a century of Hebb},
-	Crdt = {2000/12/29 11:00},
+  journal       = {Nat Neurosci},
-	Da = {20001220},
+  year          = {2000},
-	Date = {2000 Nov},
+  volume        = {3 Suppl},
-	Date-Added = {2009-04-08 18:14:47 -0400},
+  pages         = {1166},
-	Date-Modified = {2011-09-12 11:19:15 -0400},
+  month         = {Nov},
-	Dcom = {20010104},
+  issn          = {1097-6256 (Print)},
-	Edat = {2000/12/29 11:00},
+  address       = {Howard Hughes Medical Institute and Department of Brain and Cognitive Sciences, Massachusetts Institute of Technology, Cambridge 02139, USA. seung@mit.edu},
-	Fps = {Hebb, D},
+  doi    = {10.1038/81430},
-	Issn = {1097-6256 (Print)},
+  crdt          = {2000/12/29 11:00},
-	Jid = {9809671},
+  da            = {20001220},
-	Journal = {Nat Neurosci},
+  date          = {2000 Nov},
-	Jt = {Nature neuroscience},
+  date-added    = {2009-04-08 18:14:47 -0400},
-	Keywords = {21 Activity-development; 21 Cortical oscillations; 21 Neurophysiology},
+  date-modified = {2011-09-12 11:19:15 -0400},
-	Language = {eng},
+  dcom          = {20010104},
-	Lr = {20041117},
+  edat          = {2000/12/29 11:00},
-	Mh = {Animals; History, 20th Century; Humans; Long-Term Potentiation/*physiology; Memory/physiology; Nerve Net/cytology/physiology; Neuronal Plasticity/physiology; Synapses/*physiology; Synaptic Transmission/*physiology},
+  file          = {papers/Seung_NatNeurosci2000.pdf},
-	Mhda = {2001/02/28 10:01},
+  jid           = {9809671},
-	Month = {Nov},
+  jt            = {Nature neuroscience},
-	Own = {NLM},
+  keywords      = {21 Activity-development; 21 Cortical oscillations; 21 Neurophysiology},
-	Pages = {1166},
+  language      = {eng},
-	Pl = {United States},
+  lr            = {20041117},
-	Pmid = {11127829},
+  mh            = {Animals; History, 20th Century; Humans; Long-Term Potentiation/*physiology; Memory/physiology; Nerve Net/cytology/physiology; Neuronal Plasticity/physiology; Synapses/*physiology; Synaptic Transmission/*physiology},
-	Ps = {Hebb D},
+  mhda          = {2001/02/28 10:01},
-	Pst = {ppublish},
+  own           = {NLM},
-	Pt = {Biography; Historical Article; Journal Article},
+  pl            = {United States},
-	Sb = {IM},
+  pmid          = {11127829},
-	Source = {Nat Neurosci. 2000 Nov;3 Suppl:1166.},
+  pst           = {ppublish},
-	Status = {MEDLINE},
+  pt            = {Biography; Historical Article; Journal Article},
-	Title = {Half a century of Hebb},
+  sb            = {IM},
-	Volume = {3 Suppl},
+  source        = {Nat Neurosci. 2000 Nov;3 Suppl:1166.},
-	Year = {2000},
+  status        = {MEDLINE},
-	File = {papers/Seung_NatNeurosci2000.pdf},
+}
-	Bdsk-Url-1 = {http://dx.doi.org/10.1038/81430}}
 
 @article{Steriade:1993c,
 	Abstract = {As most afferent axons to the thalamus originate in the cerebral cortex, we assumed that the slow (< 1 Hz) cortical oscillation described in the two companion articles is reflected in reticular (RE) thalamic and thalamocortical cells. We hypothesized that the cortically generated slow rhythm would appear in the thalamus in conjunction with delta and spindle oscillations arising from intrinsic and network properties of thalamic neurons. Intracellular recordings have been obtained in anesthetized cats from RE (n = 51) and cortically projecting (n = 240) thalamic neurons. RE cells were physiologically identified by cortically evoked high-frequency spike bursts and depolarizing spindle oscillations. Thalamocortical cells were recognized by backfiring from appropriate neocortical areas, spindle-related cyclic IPSPs, and hyperpolarization-activated delta oscillation consisting of rhythmic low-threshold spikes (LTSs) alternating with afterhyperpolarizing potentials (AHPs). The slow rhythm (0.3-0.5 Hz) was recorded in 65% of RE neurons. In approximately 90% of oscillating cells, the rhythm consisted of prolonged depolarizations giving rise to trains of single action potentials. DC hyperpolarization increased the synaptic noise and, in a few cells, suppressed the long-lasting depolarizing phase of the slow rhythm, without blocking the fast EPSPs. In approximately 10% of oscillating neurons, the hyperpolarizing phase of the oscillation was much more pronounced, thus suggesting that the slow rhythm was produced by inhibitory sculpturing of the background firing. The slow oscillation was associated with faster rhythms (4-8 Hz) in the same RE neuron. The slow rhythm of RE neurons was closely related to EEG wave complexes recurring with the same frequency, and its strong dependency upon a synchronized state of cortical EEG was observed during shifts in EEG patterns at different levels of anesthesia. In 44% of thalamocortical cells the slow rhythm of depolarizing sequences was apparent and it could coexist with delta or spindle oscillations in the same neuron. The occurrence of the slowly recurring depolarizing envelopes was delayed by the hyperpolarizing spindle sequences or by the LTS-AHP sequences of delta oscillation. The hyperpolarization-activated delta potentials that tended to dampen after a few cycles were grouped in sequences recurring with the slow rhythm. We finally propose a unified scenario of the genesis of the three major sleep rhythms: slow, delta, and spindle oscillations.},
@@ -111410,3 +111410,5 @@ CONCLUSIONS: Centrifugal axons in the macaque retina are part of the system of a
 	Uuid = {908194C7-E3B0-4DBC-9B0A-405906B234E7},
 	Volume = {8},
 	Year = {1997}}
+
+@Comment{jabref-meta: databaseType:bibtex;}